ACINETOBACTER BAUMANNII research

For Acinetobacter, baumannii is the specific pathogen that causes Acinetobacter to form. This pathogen typically lives in contaminated water and soil. People with weakened immune systems are much more likely to have a case where an Acinetobacter is involved, seeing as there are different types, and can be cultured from patient’s saliva, urine, or other body fluids. Because Acinetobacter can cause a variety of diseases, patients with an Acinetobacter-caused infection can experience a range of symptoms from coughing and an elevated heart rate (comes with pneumonia), to low blood pressure and organ dysfunction (comes with blood or wound infection). In less severe cases, a patient may notice a fever, chills, headaches, and breathing problems (Su & Chao, 2013, p. Acinetobacter bacteria cannot be transmitted through air but there is a possibility of spreading through direct contact with the skin surfaces or objects which are already infected or contaminated by the bacteria.

Bacteria resistance can be spread in one of two ways; vertically when the bacteria are spread by the resistance genes being inherited (Perez & Bonomo, 2017, p. Or, the bacteria can be spread horizontally. This is when the bacteria are spread between different species. This can be done through what is known as conjugation. Acinetobacter is most common in those who have multiple co-morbidities and is typically harmless to those who have a healthy immune system. However Acinetobacter spp. can also multiply in open wounds of a healthy person. Symptoms of Acinetobacter infection include tachycardia, edema, low blood pressure, erythema (redness) above a wound. One of the most important features of the Acinetobacter species is antimicrobial resistance including beta-lactams, amino glycosides and quinolones.

Secondly, the samples were compared in terms of in vitro and in vivo expression. This was done using the four virulence genes out of the five colistin-resistant. Lastly, Acinetobacter baumannii samples were tasted whereby one was from the current study and four samples from the previous studies were used. Findings from the research indicated that when there is reduced expression of recA and an increase in vivo expression lpsB, dnaK, and blsA are the ones which are associated with resistance based on the isolates which were used. The difference between the isolates indicates the similarity in vitro and in vivo susceptibility when the infection caused by the drug resistant pathogen is being treated. What are the demographic and clinical characteristics and outcomes caused by CR-Aba in the Adult Intensive Care Unit.

? iii. What proportions of these CR-Aba isolates were responsible for a significant infection in the Adult Intensive Care Unit? iv. What is the prevalence of CR-Aba as 1) a colonizing bacterium and 2) a cause of clinical infection within ICU? v. What are the possible risks of infection vs. •To describe the prevalence of CR-Aba as 1) a colonizing bacterium and 2) a cause of clinical infection within ICU •To investigate possible risks of infection vs. colonization, prevalence and the outcomes of ICU patients found to have Aba cultured from a clinical specimen while staying in ICU from January 1st 2019 to December 31st 2019. It is expected that Acinetobacter baumannii will be detected in almost 10% of the samples collected from the ICU environment such furniture, gloves and beds will be the most contaminated since they are in direct contact with the patients.